CATALOGUESemaglutide
METABOLICRUO · RESEARCH USE ONLY

Semaglutide

GLP-1 Receptor Agonist

5mg / vial

Semaglutide is a long-acting GLP-1 receptor agonist and arguably the most intensively researched metabolic compound of the past decade. Its effects on appetite regulation, insulin secretion, and body weight have made it the subject of thousands of published studies spanning diabetes, obesity, cardiovascular medicine, and now neurodegeneration.

For laboratory research use only. Not for human consumption.
PRODUCT PHOTO COMING SOON

99.0%

VERIFIED PURITY — JANOSHIK

~7 days

HALF-LIFE IN RESEARCH MODELS

5mg

PER VIAL — LYOPHILISED

4113.6

G/MOL — LARGEST MOLECULE IN CATALOGUE

PRICE PER VIAL

£26.99

VERIFIED PURITY99%
LOT NUMBERZL-2024-0304
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QUANTITY
1
Subtotal£26.99
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Janoshik tested · Verify by order no.
Tracked 24 · £3.99 · Free over £100
Lyophilised · Stable in transit

What it is

Semaglutide is a 94% homologous analogue of human GLP-1 (glucagon-like peptide-1), a 30-amino acid incretin hormone secreted by L-cells of the small intestine in response to food intake. Three structural modifications distinguish it from native GLP-1: substitution of alanine with alpha-aminoisobutyric acid at position 8 (conferring resistance to DPP-IV degradation), substitution of lysine with arginine at position 34, and conjugation of a C-18 fatty diacid chain to the modified lysine at position 26 via a linker. These modifications extend its half-life to approximately 7 days and enhance albumin binding, making it the longest-acting GLP-1 analogue in research use. At 4113.6 g/mol, Semaglutide is the largest and most structurally complex molecule in the Zenic Labs catalogue.

How it works

As a GLP-1 receptor agonist, Semaglutide engages multiple overlapping mechanisms. Peripherally, it stimulates glucose-dependent insulin secretion from pancreatic beta cells, suppresses glucagon from alpha cells, and slows gastric emptying — collectively improving glycaemic control in metabolic research models. Centrally, it acts on GLP-1 receptors in the hypothalamus and brainstem to suppress appetite and reduce food intake. More recent research has identified GLP-1 receptor expression in additional brain regions, including the substantia nigra and hippocampus, driving interest in Semaglutide's potential in neuroinflammation and neurodegeneration research. Its 7-day half-life makes it practical for research protocols requiring sustained GLP-1 receptor engagement.

Research context

Semaglutide has the most extensive published research base of any compound in the Zenic Labs catalogue. Major research programmes include the SUSTAIN and PIONEER trials (glycaemic control), the STEP programme (obesity and weight reduction), and FLOW (renal outcomes). Emerging research areas include NASH/MAFLD (metabolic fatty liver disease), addiction and reward pathways, Alzheimer's disease and neurodegeneration (driven by GLP-1 receptor expression in the brain), and polycystic ovary syndrome. The breadth of active research makes Semaglutide one of the most versatile compounds available for metabolic and CNS research.

Commonly researched with

Pairs well with